Journal of Clinical and Cellular Immunology : Citations & Metrics Report
Articles published in Journal of Clinical and Cellular Immunology have been cited by esteemed scholars and scientists all around the world. Journal of Clinical and Cellular Immunology has got h-index 42, which means every article in Journal of Clinical and Cellular Immunology has got 42 average citations.
Following are the list of articles that have cited the articles published in Journal of Clinical and Cellular Immunology.
| 2024 | 2023 | 2022 | 2021 | 2020 | 2019 | 2018 | 2017 | 2016 | 2015 | 2014 | 2013 | 2012 | 2011 | 2010 | |
|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|
Total published articles |
30 | 31 | 30 | 59 | 28 | 11 | 35 | 56 | 95 | 105 | 101 | 105 | 64 | 15 | 3 |
Research, Review articles and Editorials |
2 | 13 | 8 | 23 | 18 | 8 | 25 | 35 | 54 | 86 | 79 | 35 | 23 | 14 | 3 |
Research communications, Review communications, Editorial communications, Case reports and Commentary |
20 | 18 | 22 | 22 | 10 | 3 | 10 | 21 | 39 | 6 | 9 | 7 | 1 | 1 | 0 |
Conference proceedings |
0 | 16 | 15 | 6 | 0 | 98 | 178 | 206 | 171 | 160 | 195 | 156 | 74 | 0 | 0 |
Citations received as per Google Scholar, other indexing platforms and portals |
653 | 826 | 931 | 1079 | 1018 | 971 | 871 | 851 | 753 | 550 | 276 | 145 | 43 | 40 | 0 |
| Journal total citations count | 8361 |
| Journal impact factor | 24.12 |
| Journal 5 years impact factor | 31.14 |
| Journal cite score | 30.45 |
| Journal h-index | 42 |
| Journal h-index since 2019 | 32 |
Important citations (1511)
gupta r, yadav a, aggarwal a (2016) longitudinal assessment of monocyte chemoattractant protein-1 in lupus nephritis as a biomarker of disease activity. clin rheumatol 35: 2707-14. |
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alharazy s, kong nc, mohd m, shah sa, ba’in a, et al. (2015) urine monocyte chemoattractant protein-1 and lupus nephritis disease activity: preliminary report of a prospective longitudinal study. autoimmun dis. |
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kolitz s hasson t, towfic f funt jm bakshi s, et al. (2015) gene expression studies of a human monocyte cell line identify dissimilarities between differently manufactured glatiramoids. sci rep 5: 10191. |
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hasson t, kolitz s, towfic f, laifenfeld d, bakshi s, et al. (2016) functional effects of the antigen glatiramer acetate are complex and tightly associated with its composition. j neuroimmunol 290: 84-95. |
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rovituso dm, duffy ce, schroeter m, kaiser cc, kleinschnitz c, et al. (2015) the brain antigen-specific b cell response correlates with glatiramer acetate responsiveness in relapsing-remitting multiple sclerosis patients. sci rep. |
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kolitz s, hasson t, towfic f, funt jm, bakshi s, et al. (2015) gene expression studies of a human monocyte cell line identify dissimilarities between differently manufactured glatiramoids. sci rep. |
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xu r, sun hf, williams dw, jones av, al-hussaini a, et al. (2015) il-34 suppresses candida albicans induced tnfα production in m1 macrophages by downregulating expression of dectin-1 and tlr2. j immunol res 2015. |
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vasaghi a, ashraf a, shirzadi a, petramfar p (2016) case report of lewis and sumner syndrome with bilateral vagus nerves paralysis for 16 years. american phys med rehabilit 95: e198-e201. |
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kravchenko pn (2015) mechanisms of violation of the immunological tolerance. proceedings of the karelian research center of the russian academy of sciences 12: 3-22 |
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ghotaslou r, nakhjovani m, sadeghi j, leylabadlo he, daghighazar b et al. detection of porphyromonas gingivalis dna in the synovial fluid of rheumatoid arthritis patients by real-time pcr. health sci 5: 661-665. |
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el-sayed nm, kishik sm, fawzy rm (2016) the current status of toxoplasma gondii infection among egyptian rheumatoid arthritis patients. asian pacific j trop dis 6: 797-801. |
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offer w (2014) investigating the link. |
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arleevskaya mi, kravtsova oa, lemerle j, renaudineau y, tsibulkin ap (2016) how rheumatoid arthritis can result from provocation of the immune system by microorganisms and viruses. front microbio 7. |
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arleevskaya mi, kravtsova oa, lemerle j, renaudineau y, tsibulkin ap (2016) how rheumatoid arthritis can result from provocation of the immune system by microorganisms and viruses. front microbio 7. |
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che n, li y, liu s, pan w, liu y (2015) investigation on association between five common polymorphisms in vascular endothelial growth factor and prototypes of autoimmune diseases. immunobiol 220: 722-733. |
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wang l, ding y, guo x, zhao q (2015) role and mechanism of vascular cell adhesion molecule-1 in the development of rheumatoid arthritis. exp ther med 10: 1229-1233. |
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li s, yu y, yue y, liao h, xie w, et al. (2016) autoantibodies from single circulating plasmablasts react with citrullinated antigens and porphyromonas gingivalis in rheumatoid arthritis. arthri rheumatol 68: 614-626. |
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lee jy, choi ia, kim jh, kim kh, lee ey, et al. (2015) association between anti-porphyromonas gingivalis or anti-α-enolase antibody and severity of periodontitis or rheumatoid arthritis (ra) disease activity in ra. bmc musculoskel dis 16: 190. |
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lerner a, matthias t (2015) rheumatoid arthritis–celiac disease relationship: joints get that gut feeling. autoimmun rev 14: 1038-1047. |
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comba a, çaltepe g, yank k, gör u, yüce ö, et al. (2016) assessment of endothelial dysfunction with adhesion molecules in patients with celiac disease. j ped gastroenterol nut 63: 247-252. |
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