Clinical & Experimental Cardiology
Open Access

New theory of cardiac arrhythmias in humans

9^th Annual Meeting on Arrhythmia and Cardiac Surgery

July 14-15, 2016 Brisbane, Australia

Vladimir I Ermoshkin

Russian New University, Russia

Posters & Accepted Abstracts: J Clin Exp Cardiolog

Abstract :

Objective: A new attempt to study the mechanism of arrhythmias and sudden cardiac death (SCD) analysis. Methods: Information search in the literature, participate in conferences, discussions with Russian leading cardiologists. Results: I was able to show that when considering the arrhythmia mechanisms, researchers have forgotten about the possibility of CMC excitations by mechanical pulses. Tests on the device â�?�?Cardiocodeâ�? were carried out. Under the influence of stress can be opened large arteriovenous anastomoses (AVA). Periodically, the pressure change in the arteries and veins. Venacava expands, increases tone and pulse waves start to path through the AVA on the walls of the vena cava to right heart. Mechanical impulses can excite the CMC from various points of the atria or ventricles, disrupting the sinus rhythm. The result appear extra systoles, tachycardia attacks, blocking blood flow and to any peripheral venous network sites, swelling. Longevity attacks of tachycardia and arrhythmia can lead to progressive fibrosis of the heart because of myocardial ischemia. This increases the likelihood of the onset of fibrillation and SCD. Conclusions: Unhealthy lifestyle, the presence of opening/closing AVA can sometimes lead to a lot of diseases. To get rid of attacks cardiac arrhythmias and to get SCD prevention is necessary in some way to suppress the mechanical waves, jogging on circuit: aortaartery- AVA- vein- vena cava- atria- ventricles. That is why the extra hearts beats occur with the same coupling intervals or the strictly identical periods of beats. In such cases â�?�?reentryâ�? phenomenon has a mechanical nature.

Biography :

Email: evlad48@list.ru