Journal of Clinical and Experimental Ophthalmology
Open Access
ISSN: 2155-9570
ISSN: 2155-9570
Amaghere O Charles
National Eye Centre, Nigeria
Scientific Tracks Abstracts: J Clin Exp Opthamol
Spontaneous venous pulsation is viewed as a subtle variation in the caliber of the retinal vein(s) as they cross the optic disc. The pressure gradient of the retinal vein varies because of the difference between the intraocular space and the cerebrospinal fluid. Various theories have been propounded to explain what is going on: chief amongst them is that the intraocular pressure rises and exceeds the venous pressure during systole thereby causing the veins to collapse. Retinal venous pressure exceeds IOP, making the vein to expand. Of much importance is the fact that when the intracranial pressure rises, the intracranial pulse pressure rises to equal the intraocular pulse pressure and the spontaneous venous pulsations cease. In this manner, the cessation of the spontaneous venous pulsation becomes a sensitive marker of raised intracranial pressure. The fact that pulsations are increased in patients with significantly elevated intraocular pressure, intraocular hypotension, and increased intracranial pressure has been associated with absent pulsations. Therapies that lower intraocular pressure without also lowering intracranial pressure (such as topical rather than oral carbonic anhydrase inhibitors) should be favored.
Amaghere O. Charles he is (Doctor of Optometry Nigeria), Director of Outreach Eye Plus Project (Nig.) He is Chief Medical Director (CMD) Nationale Eye Centre Abuja, Nigeria. And also President; Eye Plus Vision Africa Initiative (NGO). Dr. Amahere Charles is a Researcher, he is Mentor and International Speaker, and an Award Winner.
E-mail: drcharlesamaghere@gmail.com